Bullets in Medical Surgical Nursing

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BULLETS (Authored from previous board exam questions)  Chest X ray  painless procedure 

Bronchoscopy o AtSO4  Anticholinergic  mimics SNR  Decreases saliva  dry mouth o NPO 6 to 8 hours o Local anesthesia  check gag reflex before feeding



ABG o o o o o

o o

o

Hyperventilation  decreased CO2  increased blood pH  respiratory alkalosis Hypoventilation  increased CO2  decreased blood pH  respiratory acidosis Diarrhea  decreased HCO3  decreased blood pH  metabolic acidosis Vomiting gastric content  decreased HCL  increased blood pH  metabolic alkalosis Vomiting blood  decreased O2  anaerobic metabolism  formation of lactic acid  decreased blood pH  metabolic acidosis Blood pH  normal 7.35 to 7.45  If increased  alkalosis; If decreased  acidosis Partial CO2  normal 35 to 45  If increased Respiratory Acidosis; if decreased Respiratory Alkalosis Partial HCO3  normal 22 to 26  If increased Metabolic alkalosis; If decreased metabolic acidosis

 Cancer of the larynx  CS, alcohol and over usage of voice (choir member) o o o o



A - nterior neck mass B – urning sensation with hot beverages / Bad breath C - hange in the voice (hoarseness) D – ysphagia/dyspnea

Chronic Obstructive Pulmonary Disease o Chronic Bronchitis  Blue bloater   Excessive mucus production o Asthma  Periods of bronchospasm and bronchoconstriction o Emphysema  Disequilibrium of elastase and antielastase  Pink puffer  o Manifestations  A – LTERATION IN • LOC  decreased O2 • Thoracic anatomy  over distention of alveoli  TD = APD  barrel chest Skin • o Temperature  cool clammy skin o Color   pale to cyanotic

ABG  Respiratory acidosis  Increased CO2  B – reathing  difficulty, purse lip  expiration > inhalation  removal of excess CO2 (diet low CHO)  C – ough (mucus production); Chronic hypoxia (2 to 3 lpm of O2 therapy, decreased O2 demand by rest and SFF)  clubbing of the fingers and decreased TP to the kidneys causing polycythemia  D – ecreased Metabolism • Anorexia  weight loss (high calorie diet)  fatigue  weakness  Bronchodilators o Theophylline and aminophylline •

 

Primary effect  stimulates beta 2 receptors  smooth muscle relaxation  bronchodilation Side effect  stimulates beta 1 receptors  increases cardiac rate  need not to notify the physician

 

Adverse effect  hypotension  monitor BP  sign of toxicity Evaluation  check breath sounds



Acute Respiratory Distress Syndrome o Causes  A – spiration  R – espiratory trauma (embolism) • fracture  embolism  ARDS  D – rug toxicity (ASA)  S – epsis and shock Vomiting, bleeding, dehydration  hypovolemia  shock  ARDS • o Syndrome  Severe hypoxia  Bilateral infiltrates  Dyspnea



Pulmonary embolism o Restlessness  earliest sign



Water Seal System Drainage Bottle → marked the level every shift o o Water seal bottle  Presence of fluctuation → normal  Absence of fluctuation → lungs are fully expanded confirm) OR presence of obstruction  Intermittent bubbling → normal • Absent → obstruction • Continuous → leakage o Suction Control → continuous bubbling → normal





assess first patient (X ray

Risk factors for cardiovascular disorders R – ace  non modifiable o o I – ncreased blood pressure  modifiable o S – tress  SNR  increased BP and CR, vasoconstriction  modifiable o K – nowing sedentary life style  modifiable o F – at foods  atherosclerosis  modifiable o A – lcohol (modifiable) / Age  above 40 (non modifiable) C – igarette smoking  vasoconstriction (nicotine)  modifiable / Contraceptive pills  clotting o of blood  thrombus formation o T – ype A behavior (modifiable)  competitiveness, perfectionist  high stress level O – besity o o R – esult of DM  lipolysis  increased fatty acids  atherosclerosis o S – ex gender   males > female (before menopausal because estrogen decreases PVR) after menopausal female eversible}[inverted T wave]  Injury [elevated ST segment] > male

 Decreased TP in heart  Ischemia (Angina) {r necrosis (MI) {irreversible}[pathologic Q

wave/permanent in the ECG]  Eating a heavy meal, strenuous exercise, sex, exposure to cold  Decreased blood flow (heart) 

decreased TP (heart)  decreased O2 (heart)  anaerobic respiration  production of lactic acid PAIN  management decreased O2 demand by rest and SFF 

Angina o Pain relieved by rest and NTG o NTG  Vasodilation  orthostatic hypotension  move gradually  Monitor BP  Store in a dark and amber container 

  

Effective  tingling sensation  no need to notify physician Maximum of 3 tablets with 5 minute interval

MI o

Pain relieved by Morphine SO4  Narcotic analgesic  Can cause respiratory depression  monitor RR and O2 saturation  Antidote  narcan

 Cardioversion  synchronous  Defibrillation  unsynchronous

 Buerger’s disease  CS  vasoconstriction  stop CS  common in men  Raynaud’s  stress and cold  vasoconstriction  common in female 

Congestive heart failure Left sided  pulmonary o  Dyspnea  Crackles  Polycythemia  due to decrease O2 to the kidneys  Clubbing of the fingers  due to prolonged hyxia  Orthopnea Right sided  systemic o  Hepatomegaly  Distended neck veins  Edema  Portal hypertension  Ascites  weight gain  Varicose veins o Digoxin  Cardiac glycoside  Positive inotrophic effect  increased strength of myocardial contraction  Negative chronotrophic effect  decreased cardiac rate  monitor CR  never give if  CR below 60 bpm  Adverse effect V – omitting • • A – norexia • N – ausea • D – iarrhea A – bdominal pain • • REMEMBER: earliest  GI; late  halo vision • Antidote Digibind

 Decreased RBC

Activity in tolerance, Fatigue, provide rest, Anemia

 Decreased Platelets

Prone to bleeding, avoid parenteral injection, appl pressure on injection site, high risk for injury

 Decreased WBC  Increased WBC

prone to infection, reverse isolation presence of infection

 First Day/Newly diagnosed 

Knowledge deficit

Diuretic o D – iet  high K diet except aldactone o I – input and Output  expected increased output o U – ndesirable effect electrolyte imbalance (K)

o o o o o

R – ecord weight  expected decreased weight E – lderly  special precaution T – ake in AM and with food I – ncreased orthostatic hypotension  monitor BP and move gradually C – ancel alcohol because of mild diuretic effect

 Heparin  anticoagulant  prevent further enlargement of clot not dissolve them  monitor 

APTT/PTT  antidote protamine SO4  Coumadin  anticoagulant  prevent further enlargement of clot not dissolve it  monitor PT 

vitamin K is the antidote  Urokinase/Streptoase

dissolves the clot

 Pernicious anemia  absence of intrinsic factor (gastric surgery)  problem in absorption of Vitamin

B12  beefy red tongue schilling’s test  definitive test  24 hour urine collection  life long Vitamin B12

 Gastritis  LUQ pain  Gastric ulcer   affected area stomach  pain (precipitated by food intake  increased HCl)  pain

relieved by antacids  Duodenal ulcer   affected area duodenum  pain (2 hour after eating)  pain relieved by food  Ulcers  bleeding  (+) occult blood test (guiac)  high fiber diet, avoid red meat, iron, steroids,

NSAIDs, indomethacin  Vagotomy  resection of vagus nerve  decreased cholinergic stimulation  decreased HCl and

gastric movement  Dumping syndrome  tachycardia and weakness  3 D’s (diarrhea, diaphoresis and dizziness) 

fluids after meals, lie down after meals and SFF  Appendicitis  RLQ pain  avoid heat pads  cause rupture  signs of ruptured appendix 

sudden cessation of pain, elevation of temperature and WBC  Diverticulitis  LLQ pain  Diverticulosis



low fiber diet

high fiber diet

 Ulcerative colitis  bloody diarrhea 15 to 20 times a day  fluid volume deficit, anemia  Liver cirrhosis  alcohol and malnutrition (laennec’s), infection and drugs (post necrotic), RSCHF

(cardiac) and biliary obstruction (biliary) o Portal hypertention can lead to  Blood shifted to the different collateral Esophageal varices • Spider angioma (face and neck) • Caput medusae (abdomen) • Hemorrhoids (rectal) • Management avoid rupture  avoid shouting, valsalva maneuver  •  Increased hydstatic pressure  fluid shifting  ascites Decreased albumin  decreased oncotic / colloidal osmotic pressure  fluid shifting ascites o  management high protein diet o CHON metabolism  by product ammonia  liver cannot convert to urea  increased level of  ammonia in the brain  Alteration of LOC and changes of behavior and asterexis hepatic encephalopathy  management low CHON diet and lactulose for removal of ammonia  Hepatitis A  fecal oral  prone plumber   Hepatitis B  body secretion  prone working in a dialysis

 Cholecystitis  5 F’s (fair, female, fat, fertile and forty)  RUQ pain  after ingestion of fatty food 

demerol to relieved pain  Cholecystectomy  T tube  level of the incision site  drain excess bile  Pancreatitis  alcohol  autodigestion  LUQ pain 

Anterior Pituitary gland o Growth hormone  Increased before the closure of the epiphysis of the long bones  gigantism  tall  Increased after the closure of the epiphysis  acromegaly  big hands (big gloves), big feet (big shoes) and big head (big hat)  Decreased  dwarfism o Prolactin   o

ACTH  

o

Increased  galactorrhea Decreased  decreased milk production Increased  secondary cushing’s Decreased  secondary addison’s

TSH 

Increased  secondary hypethyroidism  Decreased  secondary hypothyroidism  Posterior pituitary gland o ADH  

Increased  water retention  oliguria  edema (fluid volume excess) and weight gain  concentrated urine  increased urine specific gravity Decreased  water excretion  polyuria  dehydration (fluid volume deficit and weight loss)  diluted urine  decreased urine specific gravity



Parathyroid gland o Parathormone  Increased  increased calcium in the blood and decrease calcium in the bones  stone formation and decreased bone mass  osteoporosis  management increased water  intake  Decreased  hypocalcemia  calcium supplement



Thyroid Gland o Increased (hyperthyroidism)  T3 and T4  increased BMR  hyperactive  inability to focus  insomia  increased catabolism  weight loss  increased appetite  increased peristalsis  Diarrhea  fluid volume deficit  Increased CR and RR (due to increased BMR) • Increased T3  heat intolerance  Calcitonin  decreased calcium in the blood  tetany  compensatory  calcium withdraws from the bones  bone destruction (complication)  PTU  decreased synthesis of TH  watch out for SE (similar to signs and symptoms of hypothyroidism)  watch out for agrunulocytosis (fever, skin rash and sore throat)  Lugol’s solution  decreased released of TH  before thyroidectomy  decreased vascularity of the thyroid gland o Decreased (hypothyroidism)  T3 and T4  decreased BMR  hypoactive  sleeps a lot  decreased metabolism  weight gain  anorexia  decreased peristalsis  constipation  decreased CR and RR due to decreased BMR  T3  cold intolerance  Calcitonin  hypercalcemia  stone formation  Synthroid and Proloid  increased TH



Adrenal Gland o Incresead (cushing’s)  Glucocorticoids  hyperglycemia and decrease wound healing



o



Mineral corticoids  increased aldosterone  sodium retention and potassium excretion  hypernatremia and hypokalemia Hypernatremia  water retention  oliguria  edema (moon face,buffalohump, • fluid volume excess and weight gain)  concentrated urine  increased urine specific gravity  low sodium diet Hypokalemia  weakness  Prominent U wave  high potassium diet •  Epinephrine and Norepinephrine  Increased BP and CR  Sex hormones • Males  gynecomastia and falling of hair  • Females  hirsutism and deepening of the voice Decreased (addisons)  Glucocorticoids  hypoglycemia and inability to cope with stress  Mineralcorticoids  decreased aldosterone  sodium excretion and potassium retention  hyponatremia and hyperkalemia • Hyponatremia  water excretion  polyuria (dehydration, fluid volume deficit and weight loss)  diluted urine --. Decreased urine specific gravity  increased fluids and Na • Hyperkalemia  weakness  tall or peaked T waves  low K diet  Epinephrine and Norepinephrine  decreased BP and CR

Diabetes Mellitus Type I  absolutely no insulin  thin  insulin o o Type II  insufficient insulin  obese  OHA o Diet  50% CHO, 30% Fats, 20% CHON Exercise  Increased uptake of glucose  Decreased insulin requirement o o Oral hypoglycemic agent (OHA)  Stimulates pancreas to produce insulin

o

Insulin 

o

SC; IV if DKA  Never massage the area  Never administer cold insulin  Rotate the site of injection PREVENTS LIPODYSTROPHY •  Mix Aspirate clear first • Inject air to cloudy first • Hypoglycemia  W – eakness  H – unger pangs  A – alteration of LOC  T – achycardia and tremors 

o o

o o

A – bdominal pain  B – blurring of vision  C – ool clammy skin  D – iaphoresis  Give  orange juice (simple sugars) DKA increased lipolysis increased ketones Hyperglycemia  polyuria, polydipsia, polyphagia, kussmaul breathing, glycosuria, ketonuria and warm flush skin Glycosylated hemoglobin  reflect BSL for the past 3 to 4 months  most accurate Foot care  Podiatrist  Avoid removing corns and calluses  Cut toe nails straight across  Avoid walking bare foot

 Hepatitis A



fecal oral

 Hepatitis B



body and bloody secretions (hemodialysis)



Peritoneal Dialysis o Diasylate output is decreased → turn patient from side to side Complication → infection → monitor WBC and temperature, diasylate is cloudy → boardlike and o rigid abdomen → peritonitis o Don’t include diasylate solution in the output of the client o Expected → decreased weight → monitor weight before and after  → decreased createnine and BUN

 Heart block 



Parkinson’s diasease o Decreased dopamine in the basal ganglia → levodopa to increased dopamine foods o Cardinals signs → tremors (non intentional) → muscle rigidity → bradykinesia o Pill rolling Microphonia → ask your client to speak aloud to be aware o o Artane and Cogentin → anticholinergic → decreased muscle rigidity



o





avoid Vit B6

Undermedication → myasthenic crisis → give cholinergics Over medication → cholinergic crisis → give ATSO4

Multiple Sclerosis o Demyelinization of the myelin sheath o Charcoat’s triad  Intentional tremors  Scanning of speech  Nystagmus o Visual disturbances → diplopia

 Pancreatitis





Myasthenia Gravis Tensilon test → confirmatory test o o Decreased Acetylcholine and increased cholinesterase Muscle weakness → priority airway o o NO tranquilizer, Morphine SO4, Muscle relaxant and neomycin Cholinergics (mestinon) → increased muscle strength → antidote ATSO4 o 



decreased tissue perfusion

autodigestion Elevated amylase →



alcohol



bleeding



shock

Rheumatoid Arthritis o No specific diagnostic test o NSAID’s and ASA (antipyretic, analgesic and anti-inflammatory) o Synovitis → Pannus formation → fibrous ankylosis (limited joint movement) (joint fixation) o Avoid flexion and promote prone position Gouty Arthritis o Increased uric acid



allopurinol and avoid organ meats (liver)

Osteoarthritis Most common → related with aging o o Pain after weight bearing exercise or activity







Bony ankylosis

tophi (ears)

rest to relieved pain



weight reduction

 Diverticulitis

LLQ pain and low fiber diet

 Cyclophosphamide (Cytoxan)

 Vincristine (Oncovin)  Iron supplement

can cause hemorrhagic cystitis



to avoid increased fluid intake

increased fiber in the diet

When is the best time to take (empty stomach), How is best taken (with orange

 juice)



Steroids and NSAID’s o DEATH inflammation o BIRTH side effects     

 Cataract



 Glaucoma

B – one marrow depression → prone to infection → monitor temperature and WBC I – ncreased gastric irritation → take it with food or after meals R – enal toxicity T – innitus H – epato toxic

common cause is aging (senile)



opacity of the lens

increased IOP → decreased of peripheral vision first → miotics (constricts pupils) → avoid ATSO4 (dilates pupil) →

 Retinal detachment



trauma



blood clots



floating spots





position on the unaffected side



halo, tunnel and gun barrel vision

dependent position → scleral buckling

 Avoid Increased Intraocular pressure o

PRIORITY Avoid vomiting, coughing, valsalva maneuver, lifting heavy objects, bending, crying

 Meniere’s o

 ASA

Triad → tinnitus, impaired hearing loss and vertigo → low Na diet Vertigo → imbalance → high risk for injury → decreased vertigo by focusing on one side of the room → assume a flat or reclining position





8th cranial nerve damage



tinnitus, impaired hearing loss and vertigo

 Antibiotics → allergic reactions



Normal Values o BUN = 10 – 20 mg/dl o Calcium = 9 to 10.5 mg/dl o Creatinine = 5 to 1.5 mg/dl o GTT = 70 to 115 mg/dl o O2 sat = 97 to 98%



Signs and Symptoms of Increased Intracranial Pressure o B – lood pressure and temperature are elevated R – espiratory and cardiac rate are decreased o o A – lteration of LOC o I – rritability o N – ote for projectile vomiting o S – eizure

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